H311 Approach to the Patient with Critical Illness

INTRODUCTION

ASSESSMENT OF ILLNESS SEVERITY

THE SOFA SCORING SYSTEM

Sepsis Gets an Upgrade With SOFA/qSOFA

THE APACHE II SCORING SYSTEM

https://image.slidesharecdn.com/icuscoringsystems12-2012postgraduateicucourse-140415151833-phpapp01/95/icu-scoring-systems-14-638.jpg?cb=1397575212

THE SAPS SCORING SYSTEM

SHOCK

INITIAL EVALUATION

FIGURE 293-2
Approach to the patient in shock.

RESPIRATORY FAILURE

TYPE I: ACUTE HYPOXEMIC RESPIRATORY FAILURE

occurs with alveolar flooding and subsequent intrapulmonary shunt physiology.

FIGURE 293-5
Pressure-volume relationship in the lungs of a patient with acute respiratory distress syndrome (ARDS). At the lower inflection point, collapsed alveoli begin to open and lung compliance changes. At the upper deflection point, alveoli become overdistended. The shape and size of alveoli are illustrated at the top of the figure.

TYPE II: HYPERCAPNEIC RESPIRATORY FAILURE

consequence of alveolar hypoventilation and results from the inability to eliminate carbon dioxide effectively.

TYPE III: LUNG ATELECTASIS

  • most common pulmonary complications in the first 72 hours after surgery
  • Patients subsequently develop regional alveolar collapse and hypoxemia from a V/Q mismatch as well as a right-to-left pulmonary shunt in the area of atelectasis.
  • To compensate for the resulting hypoxemia, the patient hyperventilates to increase ventilation of the noncollapsed alveoli and often develops respiratory alkalosis. Hypoxemia due to a right-to-left pulmonary shunt cannot be improved by 100% O2 delivery; instead, alveolar recruitment needs to be facilitated using adequate analgesia and lung expansion maneuvers.

TYPE IV: METABOLIC DEMANDS

hypoperfusion of respiratory muscles in patients in shock.

CARE OF THE MECHANICALLY VENTILATED PATIENT

MULTIORGAN SYSTEM FAILURE

MONITORING IN THE ICU

PULSE OXIMETRY

RESPIRATORY SYSTEM MECHANICS

FIGURE 293-6
Increased airway resistance with auto-PEEP. The top waveform (airway pressure vs. time) shows a large difference between the peak airway pressure (80 cmH 2 O) and the plateau airway pressure (20 cmH 2 O). The bottom waveform (flow vs. time) demonstrates airflow throughout expiration (reflected by the flow tracing on the negative portion of the abscissa) that persists up to the next inspiratory effort.

CIRCULATORY STATUS

PREVENTION OF COMPLICATIONS OF CRITICAL ILLNESS

SEPSIS IN THE CRITICAL CARE UNIT

NOSOCOMIAL INFECTIONS IN THE ICU

DEEP-VENOUS THROMBOSIS (DVT)

STRESS ULCERS

NUTRITION AND GLYCEMIC CONTROL

ICU-ACQUIRED WEAKNESS

ANEMIA

ACUTE KIDNEY FAILURE

NEUROLOGIC DYSFUNCTION IN CRITICALLY ILL PATIENTS

DELIRIUM

ANOXIC CEREBRAL INJURY

STROKE

SUBARACHNOID HEMORRHAGE

STATUS EPILEPTICUS

BRAIN DEATH

WITHHOLDING OR WITHDRAWING CARE